Human TNF-α ELISA antibody pair - 20-plate

SKU
CT747-20
Validated ELISA antibody pair with standard and conjugate; reagents available separately (1920 determinations).
Format
20-plate
Price
€ 1,055
Reactivity
Human
Category
ELISA
Analyte
TNF-α
Description
Our ELISA antibody pairs are extensively validated for high assay performance using the same reagents and materials included in U-CyTech ELISA kits. These antibody pairs are optimized to ensure reliable, sensitive, and reproducible results in a wide range of ELISA applications.
The ELISA antibody pairs are available in convenient 10- or 20-plate formats, providing 960 or 1920 determinations, respectively. Each set is supplied with either 5 or 10 vials of standards and 2 or 4 vials of Streptavidin-HRP conjugate, offering flexibility for different experimental needs and throughput requirements.
Designed for ease of use and consistent performance, these ELISA reagents support accurate quantification in immunoassays. Additional ELISA plates and reagents can be purchased separately, allowing customization of your ELISA workflow.
Description
TNF-α (Tumor Necrosis Factor Alpha) is an immune signaling protein that triggers inflammation to fight infections. It also regulates cell survival, cell death, and tissue regeneration. TNF-α is mainly produced by macrophages, and also by T cells, B cells, dendritic cells (DC) and mast cells. TNF-α is essential for normal immune responses, but when overproduced, it can contribute to inflammatory and autoimmune diseases.
Alternative names
TNF-α, TNF alpha, TNF-alpha, Tumor necrosis factor α, Tumor necrosis factor-α, Tumor necrosis factor alpha, Tumor necrosis factor-alpha, TNFA, TNF, TNFSF2, TNLG1F, DIF

References

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Mehrouz Dezfulian A new Alzheimer's disease cell model using B cells to induce beta amyloid plaque formation and increase TNF alpha expression Int Immunopharmacol 2018 59 106-112
Marta Monguió-Tortajada Low doses of LPS exacerbate the inflammatory response and trigger death on TLR3-primed human monocytes Cell Death Dis 2018 9 499